Buy Nolvadex Clenbuterol
Buy Nolvadex Clenbuterol
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When you start using anabolic roids you will probably use for years not only once. Here comes the bad part.

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Clenbuterol

Clenbuterol information:

Are you looking to buy some clenbuterol for quick weight loss in UK, you're landed at right place.

Clenbuterol is not a steroid, but a Beta 2 Sympathomitetic and central nervous system (CNS) stimulant. It is a specific agonist, stimulating the adrenergic beta 2 receptors. It is used in certain countries in a medical sense as a bronchodilator in the treatment of asthma, though not in the UK and USA, mainly due to its long half life.

Benefits of Clenbuterol

Athletes and bodybuilders use the drug due to its thermogenic and anti-catabolic effects. This is down to its ability to slightly increase the body's core temperature, thereby raising calorie (energy) expenditure. It is thought that a increase yields around a 5% increase in maintenance calories burned. Studies on livestock suggest that clenbuterol also has anabolic properties. However, this seems not to be the case in humans, thought to be due to the fact that humans lack the abundance of beta 3 receptors which increase insulin production and sensitivity.

* For fat loss . purchase clenbuterol from Anabolics2BuyUK seems to stay effective for 3-6 weeks, then it's thermogenic properties seem to subside.

* Buy Clenbuterol UK is effective in helping to burn bodyfat Clenbuterol is effective in increasing muscle mass and decreasing fat loss.

* Get Clenbuterol . it is most commonly used on patterns of 2 week on clen, 2 week off clen cycle.

PLEASE NOTE THE CLENBUTEROL IN STOCK ARE THE YANSUAN CHINA CLENBUTEROL 40mcg x 100 TABLETS

Clenbuterol UK side effects are very much dose dependent. Reported by some clen users: Headaches, hand shakes, cramps, sweating, hypertension.

Average Dosage:

Clenbuterol UK dosage: 80-140 mcgs (men)per day, divided throughout the day



Contents

Profile

Clenbuterol (often called just "Clen") is used by athletes and bodybuilders for it’s ability as a beta-2 agonist. It therefore stimulates your beta-2 receptors, which in turn help you to lose fat by allowing your body to release and burn more stored fat. Clen has been used for literally decades in the foreign veterinary world, for increasing the lean yield of livestock. It is clearly a very effective agent for this purpose, although its long half life and tendency to stay active in the body for long periods of time mean that vets in the United States aren’t able to use it. This is also the reason why (although it’s an asthma medication) it’s not available to asthmatics in the US of A. Albuterol is Clen’s shorter acting cousin, and that’s the FDA’s drug of choice here. But in the world of athletics, Clenbuterol has a much longer history of use.

Specifically, it’s used for fat loss, and since we’re talking about fat loss here, and this purpose is what it’s most often used for by athletes. Briefly stated, Clen is used as a repartitioning agent, and what this means is simply that it will increase your ratio of Fat Free Mass (FFM) to Fat Mass (FM) [ 1 ]. When you use Clenbuterol, besides (of course) noticing some fat loss, you’ll feel your body temperature rise a bit, and your appetite will be slightly repressed. [ 2 ]

Anyway, as you may have guessed, because the FDA doesn’t allow Clenbuterol use in asthmatics, and the USDA doesn’t allow it in livestock, there aren’t a lot of human studies to really examine with regards to Clenbuterol. Unfortunately this makes research a bit difficult, as it’s well known that animals have a some important differences in their beta-receptor type and concentrations, but animal studies are still quite useful here.

Clenbuterol is quite anti-catabolic and/or anabolic in almost every (animal) study ever done on it, although this hasn’t been studied or confirmed in human studies [ 3 ]. Also, a trend we see with Clenbuterol administration in animals is that the doses used are very high- more than anyone I’ve ever heard of actually taking. So, what I’m saying is that if Clenbuterol is anabolic or anti-catabolic in humans, only mild anabolic or anti-catabolic effects can realistically be expected. We can take a look at horses given a human-like dose of clen (slightly over 1mcg/lb x2 a day) and exercised for nearly human-like times (20mins, 3x a week) showed very significant decreases in %fat (-17.6%) and fat mass (-19.5%). Interestingly, this significantly increased (+4.4%) at week 6 [ 1 ]. This has been one of the reasons I have never believed in the 2 weeks on and 2 weeks theory of Clenbuterol administration. Why wouldn’t we want to use it for at least 6 weeks, considering the fact that it seems to have some profound effects during later administration. A "second wind" so to speak (get it? "second wind"? it’s an asthma med! Ha! Ok…moving along…).

One of the primary drawbacks of Clenbuterol is that after a couple of weeks, it seems to stop working for most people. This is because it can cause a downregulation of pulmonary, cardiac and central nervous system beta-adrenergic receptors [ 4 ]. This is why it seems to stop burning fat for most people at that point. To counteract this, you can take some Ketotifen, Benadryl, or Periactim every 3rd or 4th week that you remain on Clenbuterol. These are prescription anti-histimines, so they’ll make you drowsy (take before bedtime).

Also, bear in mind that clen isn’t great for your heart, and can cause some issues there (enlargement of ventricles, etc…) but most studies showing Clen to cause heart problems are with animals, and even though the dosing is almost similar to what humans take (in some studies its within range of what would be double of a large human dose. ). Again, it’s important to remember that animals have more beta-2 receptors and they cause certain event chains that humans’ beta-2 receptors may not, due to their relatively high concentrations. Clen causes cardiac hypertrophy to some degree, in some cases and even dose-dependent apoptotic and necrotic myocyte death [ 5 ]. And since Clen depletes taurine [ 6 ] as do most if not all beta-agonists, you may want to supplement your Clen use with some Taurine.

One of the weirdest things about Clenbuterol is that even though it’s an asthma medication, studies have shown reduced exercise (cardiovascular) performance with Clen [ 7 ]. but some also show that Clen can alleviate exercise induced asthma [ 8 ] !

Clenbuterol is one of the easiest drugs to find proper dosing for, and I’ve always made the same recommendations as to finding the appropriate dose for you. Basically, start with 20mcgs upon rising. If the side effects (possible anxiety, and shaking or sweating) aren’t too pronounced, then repeat that same dose again later in the day, and then once again in that day (again, if you find you can tolerate the effects). If you start experiencing intolerable sides, then decrease the does to where it’s tolerable. If not, then start increasing the dose more, very gradually.

Don’t go over 200mcgs, though…and keep your Blood Pressure at (or under) 140/90. If your Blood Pressure goes over that, reduce your dose. If side effects are intolerable, decrease your dose.



Thread: Taking clenbuterol with nolvadex (as a pct - but more for fat loss/shredding)

Taking clenbuterol with nolvadex (as a pct - but more for fat loss/shredding)

hey,

Im starting a cycle of nolvadex of 40mg a day and i will be taking clenbuterol for 2 weeks on, two weeks off (for 6 weeks).

Im trying to shred this fat of my gut annoying lol, i know theres better options out there for shredding, i want some advidce towards best way to take clenbuterol?

My training program (60min weights - probably 60/80% max for 10/12 reps to build definition) then a 60 min cardio session, then a 5 min session of tabata. Thats just a brief out line of my routine, im cutting the fat intake down to help with results.

(all training starts around 5pm)

Im have 100 x 40mcg tabs of clenbuterol, but as it can affect respiritarory i want to know when the best time to take this is?

Any help is much appreciated.



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Clenbuterol

Overview and History of Clenbuterol

Clenbuterol is not an anabolic steroid. but instead belongs to a certain class of compounds and drugs known as sympathomimetics (more commonly known as stimulants). It is specifically a sympathomimetic amine. Many other drugs belong to this ‘family’, some of which include: Caffeine, Albuterol, Ephedrine, Dextroamphetamine, Cocaine, and many others. This is a very broad drug category. All of these compounds, which include Caffeine, Albuterol, Clenbuterol, Ephedrine, Epinephrine, Norepinephrine, etc. are all related to one another and could be considered siblings or ‘cousins’ to each other, considering they are related in many ways and carry many similarities between one another. Clenbuterol in particular stimulates the sympathomimetic central nervous system in a few different ways. These nervous system stimulating effects work through the manner of receptors known as adrenoreceptors. These receptors are found in a plethora of different tissues throughout the body. Among many of the tissues these receptors are located, Clenbuterol acts upon these receptors and the result manifests in the form of different effects depending on which type of cells the receptors are located on. It is important to understand that there exist approximately 9 different adrenoreceptors throughout the body. As such, they are labeled as Alpha or Beta receptors with a series of numbers attributed to them for further labeling purposes. For example, there are Alpha-1, Alpha-2, Beta-1, Beta-2, etc. receptors located throughout the body. The different stimulants and sympathomimetic compounds differ in not only their individual action that they may have over their cousin compounds, but also in how they interact with these receptors. Its specifically well known for its interaction with the Beta-2 receptors, which is why its referred to as a Beta-2 receptor agonist. The relevance of this to Clenbuterol’s fat burning effects will be expanded upon shortly. Clen itself possesses a half-life of approximately 36 – 48 hours, which easily allow for single daily doses to be sufficient and no need for multiple daily doses.

Common Clenbuterol Uses

Even though Clenbuterol is utilized first and foremost as a fat burning agent in athletic and entertainment circles, its original use in medicine was actually very different. All kinds of stimulants and sympathomimetics have been provided a medical purpose in the treatment of a broad spectrum of medical conditions, while many of them share similar attributes and can treat some of the same medical conditions as well. Clenbuterol specifically has been used a great deal to treat individuals suffering from asthma. This is the reason why its been the primary active ingredient in asthma inhalers (although for a long time now in North America, Albuterol is now the active ingredient in use in asthma inhalers – Clenbuterol is no longer used in North America for this purpose). The reason why its so useful in treating asthma is because, upon activating Beta-2 receptors in certain tissues (specifically, the cells lining the bronchial tube pathways), it will enable bronchial dilation (expanding of the airways) in the lungs, nose, and throat. This is one of the previously mentioned attributes that all sympathomimetics share with one another (bronchial dilation) to varying degrees. The body’s own naturally created neurotransmitters Epinephrine (Adrenaline) and Norepinephrine (Noradrenaline), which are also sympathomimetic stimulants. exhibit this effect as well but to a far greater extent. Other approved medical uses for Clen include: hypertension. cardiovascular shock/slowdown, arrhythmias, migraine headaches, allergic reactions and swelling, histamine reactions, and anaphylactic shock. It has been previously mentioned that Clenbuterol is a Beta-2 agonist because of its primary action on Beta-2 receptors in the body, but it must be clarified that Clenbuterol does exhibit activity on other receptor sites, but to a far lesser degree and as a result, far less significant. As an example, when Clenbuterol is compared to Ephedrine, we see that it acts primarily on Beta-2 receptors while Ephedrine has been shown to act on a plethora of Beta and Alpha receptors equally. For this particular scenario, the following analogy for ease of explanation could be used: Clenbuterol’s action of selectively activating Beta-2 receptors is the equivalent of having several nails sticking out of a wooden surface, and a hammer is used to hammer one specific nail on the head, while Ephedrine is the equivalent of using a larger sledgehammer to hit multiple nails on the head to drive them into the wood. Although not a perfect analogy, this explains Clenbuterol’s activity with a fair amount of accuracy.

The Activity of Clenbuterol In Tissues

Clenbuterol’s use as a bronchodilator for asthma sufferers has been explained above, including its medical application for other medical conditions. What is yet to be explained, and what the main focus here is, is its effect on fat metabolism. It is a very well-known fact that it has minor anabolic effects in muscle tissue. Some animal studies have been performed which have clearly demonstrated Clenbuterol’s anabolic activity in muscle tissue, but this has been found to be a very mild and minor effect. The truth of the matter is that the studies that have been conducted, as well as the anecdotal evidence from human users and bodybuilders, have demonstrated that Clenbuterol’s anabolic effects are very negligible at best. One study conducted on Clen observing its effects on skeletal muscle tissue in rats had concluded that Clenbuterol does exhibit minor anabolic effects in animals, but that a long duration of action appears to be required in order to induce these anabolic effects[1]. It would then be advisable to any potential user of Clenbuterol to understand that anyone seeking to use it purely for its supposed anabolic effects would more than likely result in disappointment and disappointing results. If used for the purpose of physique/performance enhancement, should be regarded first and foremost as a fat loss agent and should be used as such instead of an ‘anabolic’ compound (of which it does a very poor job). Further evidence for the negligible anabolic effects come from analysis of the many studies performed on animals in which the animal test subjects had been administered Clen at doses considered extreme and highly dangerous for a human. The fact that the majority of these studies have been performed on animals has raised the question as to whether or not Clen  is actually anabolic at all in humans.

In any case, the selective Beta-2 action is what provides some significant fat loss attributes. It is through the activation of these B2 receptors located on the surface of fat cells where Clenbuterol can directly activate and initiate fat metabolism, and initiate the process of triglyceride breakdown into free fatty acids. B2 receptors on fat cells are known to be primary influences in the role of fat metabolism, and perhaps to a greater extent than other Beta or Alpha receptors located on fat cells as well. It is the enhanced rate of fat metabolism provided by Clen that is the reason why Clen buterol in the past had frequently been administered to cattle in conjunction with various anabolic steroids. The purpose of this practice was to ensure the largest amount of lean fat-free meat for slaughter and subsequently provided as food. One important factor to note for any potential user is that Clenbuterol’s fat loss effects may not be as effective in every individual. Some may respond more favorably to the fat loss effects, while others may find that they respond better in terms of fat loss with other compounds such as Ephedrine (perhaps due to Ephedrine’s greater activity with more receptor types). The point here is to remember that although Clenbuterol should elicit favorable fat loss in most individuals, non-responders (or people who respond very poorly) do exist.

One final note on Clenbuterol’s Beta-2 agonistic activity is that of Beta-2 receptor downregulation. It is through regular daily use of Clen that Beta-2 receptors can and will down regulate in response to Clenbuterol’s activity on those receptors[2]. What results is a continually diminished fat loss effect over time, which must be remedied one of two different ways. The first method would be time off from the drug (2 weeks minimum or longer). However, studies have found that the use of Ketotifen (Ketotifen Fumarate, an anti-histamine drug) actively up-regulates these Beta-2 receptors[3]. This will be further expanded upon, but diminishing fat loss resulting from the continued use of Clenbuterol tends to appear around the 2 nd or 3 rd week, and varies depending on the individual. If not using Ketotifen, a 2 week break is the typical protocol in order to allow the body to naturally up-regulate its receptor sites. Finally, what must be noted is the rumor of Benadryl (Diphenhydramine Hydrochloride) use for receptor up regulation. This is known as a false rumor, and it is not advised that any individual utilize Benadryl with the hopes of up regulating Beta-2 receptors to increase fat loss. There is no harm in the use of Benadryl with Clen, but one would be wasting their time and money engaging in this practice, and it is important to be clear that the use of Benadryl for this purpose was found to be a baseless rumor, and an ineffective practice.

Clenbuterol (AKA Spiropent, Ventipulmin)

Chemical Name:  (RS)-1-(4-Amino-3,5-dichlorophenyl)-2-(tert-butylamino)ethanol

Molecular Weight:  277.19 g/mol

Formula:  C12H18Cl2N2O

Original Manufacturer:  N/A

Half Life:  36 – 48 hours

Detection Time:  4 – 5 days

Anabolic Rating:  N/A

Androgenic Rating:  N/A

Clenbuterol References:

[1]Anabolic effects of clenbuterol on skeletal muscle are mediated by beta 2-adrenoceptor activation. Choo JJ, Horan MA, Little RA, Rothwell NJ. Department of Physiological Sciences, University of Manchester Medical School, United Kingdom. Am J Physiol. 1992 Jul;263(1 Pt 1):E50-6.

[3]Effects of ketotifen and clenbuterol on beta-adrenergic receptor functions of lymphocytes and on plasma TXB-2 levels of asthmatic patients. Huszar E, Herjavecz I, Boszormenyi-Nagy G, Slapke J, Schreiber J, Debreczeni LA. Z Erkr Atmungsorgane. 1990;175(3):141-6.
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